Method for 3D modelling based on structure from motion processing of sparse 2D images

A method based on Structure from Motion for processing a plurality of sparse images acquired by one or more acquisition devices to generate a sparse 3D points cloud and of a plurality of internal and external parameters of the acquisition devices includes the steps of collecting the images; extracting keypoints therefrom and generating keypoint descriptors; organizing the images in a proximity graph; pairwise image matching and generating keypoints connecting tracks according maximum proximity between keypoints; performing an autocalibration between image clusters to extract internal and external parameters of the acquisition devices, wherein calibration groups are defined that contain a plurality of image clusters and wherein a clustering algorithm iteratively merges the clusters in a model expressed in a common local reference system starting from clusters belonging to the same calibration group; and performing a Euclidean reconstruction of the object as a sparse 3D point cloud based on the extracted parameters

Alternância de hospedeiros como mecanismo de sobrevivência das cigarrinhas Dilobopterus costalimai e Oncometopia facialis (Hemiptera: Cicadellidae), vetoras da Clorose Variegada dos Citros (CVC)

Dilobopterus costalimai (Young) e Oncometopia facialis (Signoret) são duas das mais importantes espécies de cigarrinhas dos citros, transmissoras da bactéria Xylella fastidiosa causadora da doença conhecida como Clorose Variegada dos Citros (CVC). Com o objetivo de se desenvolver uma técnica de criação destas espécies, em condições de laboratório, estudou-se a preferência por postura e o desenvolvimento ninfal, em diferentes sistemas de criação, tendo como plantas hospedeiras limão cravo (Citrus limonia) e falso boldo (Vernonia condensata). O delineamento experimental foi em blocos casualizados com três tratamentos e oito repetições. Fêmeas de D. costalimai tiveram acentuada preferência para ovipositar em folhas de limão cravo, enquanto que fêmeas de O. facialis colocaram maior número de ovos em falso boldo, embora não diferisse estatisticamente de limão cravo. A viabilidade ninfal da espécie D. costalimai foi nula em limão cravo e de 58% em de falso boldo. Para a espécie O. facialis a viabilidade ninfal foi de 25 e 78%, em limão cravo e falso boldo, respectivamente, confirmando ser o falso boldo uma planta hospedeira mais adequada para a criação das duas espécies de cigarrinhas dos citros. Na natureza, a alternância de hospedeiros parece ser um importante mecanismo de sobrevivência das espécies vetoras da CVC.Dilobopterus costalimai (Young) and Oncometopia facialis (Signoret) are two of the most important species of citrus leafhoppers, vectors of bacterium Xylella fastidiosa which causes the Citrus Variegated Chlorosis (CVC) disease. To develop a rearing technique for these species under laboratory conditions, the egg laying preference and nymph development were studied in different breeding systems: Rangpur lime (Citrus limonia) and "falso boldo" (Vernonia condensata) as host plants. Trials were set up in a randomized block design with three treatments (n=8). Females of D. costalimai had particular preference for ovipositing on Rangpur lime leaves while O. facialis females placed a higher number of eggs on "falso boldo", but it did not differ statistically from the Rangpur lime. The nymphal viability of D. costalimai was null in Rangpur lime and 58% in "falso boldo". For O. facialis the nymphal viability was 25 and 78% in Rangpur lime and "falso boldo", respectively. "Falso boldo" is more suitable as a host plant to rear the two species of citrus leafhoppers. The alternation of host plants seems to be an important survival mechanism of the CVC-vector species, as shown in natural conditions

A programmable, multi-format photonic transceiver platform enabling flexible optical networks

Development of programmable photonic devices for future flexible optical networks is ongoing. To this end, an innovative, multi-format QAM transmitter design is presented. It comprises a segmented-electrode InP IQ-MZM to be fabricated in InP, which can be directly driven by low-power CMOS logic. Arbitrary optical QAM format generation is made possible using only binary electrical signals, without the need for high-performance DACs and high-swing linear drivers. The concept enables a host of Tx-side DSP functionality, including the spectral shaping needed for Nyquist-WDM system concepts. In addition, we report on the development of an optical channel MUX/DEMUX, based on arrays of microresonator filters with reconfigurable bandwidths and center wavelengths. The device is intended for operation with multi-format flexible transceivers, enabling Dense (D)WDM superchannel aggregation and arbitrary spectral slicing in the context of a flexible grid environment

O uso do jogo computacional para o ensino da tabela periódica no ensino de Química para alunos da EJA

Orientador : Roberto de Fino BentesArtigo (especialização) - Universidade Federal do Paraná, Setor de Educação Profissional e Tecnológica, Curso de Especialização em Mídias Integradas na Educação.Inclui referência

Blind Adaptive Chromatic Dispersion Compensation and Estimation for DSP-Based Coherent Optical Systems

We propose an accurate and low-complexity blind adaptive algorithm for chromatic dispersion (CD) compensation and estimation in coherent optical systems. The method is based on a Frequency Domain Equalizer (FDE), a low complexity Time Domain Equalizer arranged in a butterfly structure (B-TDE) and an Optical Performance Monitoring (OPM) block in a loop configuration. The loop is such that, at each iteration, the CD value compensated by the B-TDE and estimated by the OPM is given to the FDE; according to this estimation, in the subsequent iteration, the FDE compensates also this quantity. The procedure is repeated until the majority of CD is compensated by the FDE and a small residual quantity is compensated by a low complexity B-TDE with a small number of taps. The method is extended to long haul uncompensated links exploiting the information on the mean square error (MSE) provided by the B-TDE. The proposed algorithm is then experimentally validated for a polarization multiplexed quadrature phase shift keying (PM-QPSK) signal at 112 Gbit/s propagating along 1000 km of uncompensated Z PLUS® optical fiber. A statistical analysis of the performance of the proposed solution, in terms of mean value and standard deviation of the CD estimation error, is carried out, running a set of simulations including different impairments, such as noise, polarization dependent loss, polarization mode dispersion and self-phase modulation in a line of 1000 km of uncompensated G.652 optical fiber. Our method could be used to compensate and estimate any CD quantity without increasing the number of taps in the B-TDE and exploiting devices already included in the system (TDE, FDE and OPM) arranged in a loop

Tyr78Phe Transthyretin Mutation with Predominant Motor Neuropathy as the Initial Presentation

Transthyretin (TTR) amyloidosis, the most frequent form of hereditary amyloidosis, is caused by dominant mutations in the TTR gene. More than 100 mutations have been identified. Clinical manifestations of TTR amyloidosis are usually induced by extracellular amyloid deposition in several organs. The major neurological manifestation is motor-sensory neuropathy associated with dysautonomic impairment. Here, we describe a 63-year-old man who came to our institution due to a suspected motor neuron disease. During a 4-year follow-up period, he underwent extensive clinical examination, electromyographic studies, sural nerve biopsy and TTR gene analysis by direct sequencing. Despite the predominant motor involvement, the detailed clinical examination also showed some mild sensory and dysautonomic signs. In addition, his clinical and family history included multiorgan disorders, such as carpal tunnel syndrome, as well as conditions with cardiac, renal, eye, and hepatic involvement. The sural nerve biopsy disclosed amyloid deposition, and the sequence analysis of the TTR gene detected a heterozygous Tyr78Phe substitution. The TTR gene variant found in our patient had only been described once so far, in a French man of Italian origin presenting with late-onset peripheral neuropathy and bilateral carpal tunnel syndrome. The predominant motor involvement presented by our patient is an uncommon occurrence and demonstrates the clinical heterogeneity of TTR amyloidosis

Olfactory Function in Patients with Inflammatory Bowel Disease (IBD) Is Associated with Their Body Mass Index and Polymorphism in the Odor Binding Protein (OBPIIa) Gene

Smell strongly contributes to food choice and intake, influencing energy balance and body weight; its reduction or loss has been related to malnutrition problems. Some patients with inflammatory bowel disease (IBD), mainly Crohn’s disease (CD) and ulcerative colitis (UC), are underweight, while others are overweight. Some studies suggest that changes in eating habits could be linked to specific disorders of the olfactory functions. We assessed the olfactory performance in 199 subjects (healthy control (HC) n = 99, IBD n = 100), based on the olfactory Threshold, Discrimination and Identification score (TDI score), measured with the “Sniffin’ Sticks” test. Subjects were genotyped for the rs2590498 polymorphism of the OBPIIa gene. IBD patients showed both a slightly, but significantly, lower olfactory function and a higher BMI compared to HC subjects. Threshold (in both population) and Discrimination (in IBD patients) olfactory score were affected by the OBPIIa genotype. BMI was influenced by both health status and OBPIIa genotype. A lower olfactory function may delay the satiety sensation and thus increase meal duration and body weight in IBD patients. However, the AA genotype of the OBPIIa seems to “protect” IBD patients from more severe olfactory dysfunction

Progressive myoclonus epilepsies due to SEMA6B mutations. New variants and appraisal of published phenotypes

Variants of SEMA6B have been identified in an increasing number of patients, often presenting with progressive myoclonus epilepsy (PME), and to lesser extent developmental encephalopathy, with or without epilepsy. The exon 17 is mainly involved, with truncating mutations causing the production of aberrant proteins with toxic gain of function. Herein, we describe three adjunctive patients carrying de novo truncating SEMA6B variants in this exon (c.1976delC and c.2086C > T novel; c.1978delC previously reported). These subjects presented with PME preceded by developmental delay, motor and cognitive impairment, worsening myoclonus, and epilepsy with polymorphic features, including focal to bilateral seizures in two, and non-convulsive status epilepticus in one. The evidence of developmental delay in these cases suggests their inclusion in the “PME plus developmental delay” nosological group. This work further expands our knowledge of SEMA6B variants causing PMEs. However, the data to date available confirms that phenotypic features do not correlate with the type or location of variants, aspects that need to be further clarified by future studie

A new case of limb girdle muscular dystrophy 2g in a greek patient, founder effect and review of the literature

Limb girdle muscular dystrophy (LGMD) type 2G is a rare form of muscle disease, described only in a few patients worldwide, caused by mutations in TCAP gene, encoding the protein telethonin. It is characterised by proximal limb muscle weakness associated with distal involvement of lower limbs, starting in the first or second decade of life. We describe the case of a 37-year-old woman of Greek origin, affected by disto-proximal lower limb weakness. No cardiac or respiratory involvement was detected. Muscle biopsy showed myopathic changes with type I fibre hypotrophy, cytoplasmic vacuoles, lipid overload, multiple central nuclei and fibre splittings; ultrastructural examination showed metabolic abnormalities. Next generation sequencing analysis detected a homozygous frameshift mutation in the TCAP gene (c.90_91del), previously described in one Turkish family. Immunostaining and Western blot analysis showed complete absence of telethonin. Interestingly, Single Nucleotide Polymorphism analysis of the 10 Mb genomic region containing the TCAP gene showed a shared homozygous haplotype of both the Greek and the Turkish patients, thus suggesting a possible founder effect of TCAP gene c.90_91del mutation in this part of the Mediterranean area. (C) 2018 Elsevier B.V. All rights reserved.Peer reviewe